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The Difficult Airway
Careful preoperative assessment is essential including the
evaluation of:
Intubation in some congenital syndromes associated with
difficulty such as Pierre Robin becomes easier as the child ages, whilst
intubation in other syndromes such as Treacher Collins becomes progressively
more difficult as time passes. Examination of any previous anaesthetic records
relating to intubation is mandatory.
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All sedative premedication must be avoided in the presence of upper airway
obstruction.
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Inhalational induction with either sevoflurane
or halothane is often safest
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use of CPAP in the early stages of
anaesthesia is beneficial. CPAP should be maintained and IV access should then
be established whilst anaesthesia is deepened.
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once a sufficient plane of
anaesthesia has been achieved an oropharyngeal airway can be inserted. This
may be extremely useful in gaining control of the airway but should not be
attempted whilst the child is still "light" as laryngospasm may be
precipitated.
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Wide range of ETT plus gum elastic bougies and introducers should be available,
as well as a choice of straight and curved bladed laryngoscopes. The various
strategies for dealing with difficult intubations are similar to those used in
adults.
Tonsillectomy in Obstructive Sleep Apnoea
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Occasionally lymphoid hyperplasia of the tonsils may result
in obstructive sleep apnoea.
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Affected children often:
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Post tonsillectomy
complications, including apnoea, are higher in such children.
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The child should
have pre-operative sleep studies to detect periods of apnoea of hypopnoea.
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All
sedative premedication agents are contraindicated in these children.
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As
post-operative complications are higher in this group of patients, they should
be closely monitored, including pulse-oximetry, after operation.
The Bleeding Tonsil
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Hypovolaemia due to blood loss is often underestimated
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Circulating volume should be
restored and any severe anaemia corrected to restore normal cardiovascular
indices preoperatively. Bleeding is rarely so rapid that complete restoration of blood volume
can not be achieved.
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Further blood for transfusion should be readily available
in case of need and a coagulation screen performed as occasionally the child may
have an undiagnosed bleeding diathesis.
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The anaesthetic chart from the original
tonsillectomy should be examined and a note made of the size of ETT used and of
any anaesthetic difficulties encountered. It may only be possible to use an ETT half
a size smaller than the original due to generalised oedema.
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There may be little blood visible, but much may have been
swallowed. Blood is a gastric irritant and may precipitate vomiting upon
induction of anaesthesia, at the very least regurgitation of gastric contents
should be anticipated.
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Both an inhalational induction and a rapid sequence
induction are acceptable, bearing in mind that the bleeding point will be supra
glottic and thus the child can still aspirate if cricoid pressure is used.
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If an
inhalational induction is planned then it should be performed in the left
lateral, head down position to decrease the risk of aspiration.
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Suction must be
readily available at all times.
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Stomach should be emptied of blood via a
gastric tube prior to extubation and the child extubated fully awake once all
the protective laryngeal reflexes have returned.
Acute Upper Airway Problems
Signs of airway obstruction include the presence:
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stridor
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inspiratory stridor suggests an extrathoracic lesion e.g. laryngomalacia
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expiratory stridor suggests an intrathoracic lesion e.g. tracheomalacia.
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biphasic stridor is suggestive of a "fixed" lesion such as subglottic
stenosis.
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co-existing hoarseness suggests laryngeal pathology.
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recession of the tracheal, intercostal, supraclavicular and
subcostal areas
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tachypnoea
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tachycardia
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flaring of the alae nasi
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grunting
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generalised restlessness, cyanosis and an impaired ability to feed.
Causes of Upper Airway Obstruction:
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Site Neonate/Young Infant Older Child |
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Congenital |
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Nose Choanal Atresia |
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Pharynx Pierre-Robin Anomalad Macroglossia
Cystic Hygroma Mucopolysaccharidoses
(e.g. Hurlers)
Rare Syndromes with Cranio-Facial Deformity |
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Larynx Laryngomalacia Papillomata
..Congenital Subglottic Stenosis, Webs, Cysts, and
Haemangiomata..
…………….….Congenital Vocal Cord Palsy……….. |
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Trachea Tracheomalacia Vascular Rings |
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Acquired |
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Nose Nasal Congestion
Naso-gastric Tube |
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Pharynx ………………..…Thermal and Chemical
Burns…….. |
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Retropharyngeal Abscess
Angioneurotic Oedema
Enlarged Tonsils &
Adenoids |
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Larynx ….…………….…Laryngospasm…………………….
.......…...………..Acquired Vocal Cord Palsy………
CROUP
Epiglottitis
Post Intubation Oedema
Sub-glottic Stenosis,
Webs and Granulations
Foreign Body
Diphtheria |
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Trachea Foreign Body
Bacterial Tracheitis
Mediastinal Tumours |
Epiglottitis
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Due to bacterial infection, traditionally by
Haemophilus influenza b, although the incidence has fallen dramatically
following the introduction of Hib vaccine.
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Classically the child has:
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marked
fever of short duration
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inability to swallow saliva and hence drooling is seen
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adopts an open- mouthed sitting position.
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All ages can be affected but it is
commonest in the 3-6 year olds.
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Urgent intubation to secure the airway is required.
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Halothane/sevoflurane in oxygen is the safest technique in the presence of
airway obstruction and should be started with the child in the sitting position.
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Main risk is complete airway obstruction, which may develop at a
light stage of anaesthesia (too light to tolerate an airway or ETT.) Thus there
should be an ENT surgeon scrubbed on standby in case an emergency tracheostomy is needed.
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Once asleep, anaesthesia
should be deepened and CPAP applied with a tight fitting mask (Rendell-Baker
masks are not usually satisfactory.) Intravenous access should then be secured
and blood cultures taken as well as fluid +/- IV atropine given.
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Child
should intubated deep under direct vision with an oral ETT and without the use
of muscle relaxants. The epiglottis looks like a red cherry and bubbles through
the saliva during respiration may be the only clue to the location of the
larynx.
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ETT can be changed to a nasal tube once airway control is achieved.
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Muscle relaxants may only be used once the airway is secured.
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ETT much
smaller than predicted from age.
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Treatment
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antibiotics (3rd generation
cephalosporin +/- erythromycin)
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ventilation until an audible
leak around the ETT is heard and the temperature has settled, usually within
24-36 hours.
Do
NOT:
May provoke
life-threatening complete airway obstruction.
Laryngotracheobronchitis (Croup)
Clinical features
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Longer history than epiglottitis
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Typically presents in the 2nd
year of life.
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Prodromal URTI is common
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Inspiratory and expiratory
stridor with the classical "barking" cough.
Treatment
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Steroids
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Humidified oxygen
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+/- nebulised adrenaline (1ml/kg 1:1000
adrenaline, max 5ml, stop if heart rate exceeds 200bpm or dysrhythmias are
seen).
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Occasionally intubation is required
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management of anaesthesia is
as for epiglottitis, although IV access will usually have been obtained
pre-operatively.
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extubate once there is an audible leak around the ETT,
usually after 4-5 days
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antibiotics are only indicated in the presence of
secondary bacterial infection.
Removal of an Inhaled Foreign Body
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Most common indication for bronchoscopy in
children aged 1-3 years.
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Peanuts are the most commonly inhaled objects in the
UK; they are particularly dangerous as oil in the nut produces mucosal
irritation, oedema and often a severe pneumonitis distal to the obstruction.
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Typically there is a history of choking followed by coughing, stridor, and
occasionally dyspnoea or cyanotic attacks.
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Presentation is usually immediately
after aspiration but in up to 30% it may be delayed for more than 1 week.
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On
examination there may be unilateral decreased air entry or wheezing.
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CXR: foreign body may be visible and distal pneumonia or emphysema (due to
ball-valve effect) may be present. Both inspiratory and expiratory films should
be taken.
Anaesthesia
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atropine pre-med, 20m
g/kg intramuscularly should be given. IM is needed as this will ensure
that the airway mucosa is sufficiently dry for topical lignocaine to work (it
won't anaesthetise wet mucosa).
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induce with sevoflurane/halothane in oxygen and
aim for deep anaesthesia.
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topical lignocaine (max 4mg/kg) to anaesthetise
the airway, including the vallecula as this is were the surgical gag will be
placed, to prevent coughing/laryngospasm upon surgical instrumentation.
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maintain
spontaneous ventilation with the Ayre's T-piece connected to the side arm of the
ventilating Storz bronchoscope, although assisted ventilation may be necessary.
Procedure may be prolonged.
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biggest danger comes when the foreign
body is withdrawn into trachea. May fall out of the forceps, lodging just
below cords and totally obstructing airway. It is safest to push it back down
the airway sufficiently far to allow adequate ventilation and then make a second
attempt at removal. Monitor air entry to both lungs as the surgeon may dislodge
the foreign body from one side to the other.
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good communication between the
surgeon and anaesthetist is vital!
Post-operative care
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Dexamethasone (0.25 mg/kg IV and then 3 doses of 0.1
mg/kg IV 6 hourly - Great Ormond Street regime) is advisable if there has been repeated
instrumentation of the airway with the bronchoscope.
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Humidification of air with
or without oxygen and regular physiotherapy post-operatively is essential.
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Antibiotics should be administered if infection is present.
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